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详细书目
| 文件类型: | 文章 |
|---|---|
| 所有的著者/提供者: | Donald C Jackson |
| ISSN: | 0003-1569 |
| OCLC号码: | 481400174 |
| 语言注释: | English |
| 奖励: |
摘要:
During prolonged experimental submergence, freshwater turtles become anoxic and develop a combined respiratory and non-respiratory acidosis. Anoxia and acidosis are known to depress cardiac function in turtles and other vertebrate species. In vitro studies of ventricular and atrial tissue of turtles indicate that increased extracellular Ca<sup>++</sup> concentration can reverse these depressant actions. Intact turtles utilize this compensatory mechanism during anoxia and other conditions leading to acidosis by increasing plasma Ca<sup>++</sup> concentration. In addition, cardiac cells may release Ca<sup>++</sup> from cell organelles to compensate for induced respiratory acidosis. Both mechanisms presumably improve cardiac contractility by elevating the level of sarcoplasmic Ca<sup>++</sup>.
