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The placenta and neurodisability

Author: Philip Baker; Colin Sibley
Publisher: London : Mac Keith Press : Distributed by Cambridge University Press, 2006.
Series: Clinics in developmental medicine, no. 169.
Edition/Format:   eBook : Document : EnglishView all editions and formats
Summary:
Contains detailed accounts of placental lesions that affect normal structure and function to show how impaired placental function may contribute to deviant fetal growth and altered brain development.
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Genre/Form: Electronic books
Additional Physical Format: Print version:
Placenta and neurodisability.
London : Mac Keith Press : Distributed by Cambridge University Press, 2006
(DLC) 2006279560
(OCoLC)65223124
Material Type: Document, Internet resource
Document Type: Internet Resource, Computer File
All Authors / Contributors: Philip Baker; Colin Sibley
ISBN: 9781898683766 189868376X
OCLC Number: 840705364
Description: 1 online resource : illustrations.
Contents: The placenta and neurological outcome in the child / Karin Nelson --
Deficient trophoblast invasion, placental and fetal compromise / Fiona Lyall --
The materno-fetal inflammatory response / Mark Turner --
Cytokines, growth factors, placental insufficiency and infection / Ian Crocker --
Placental lesions and neurological outcome / Raymond Redline --
Placental pathology, intrauterine growth restriction, and subsequent child development / Paul Eunson --
MDR-1 P-glycoprotein in the placenta: a protective role in neurodevelopment? / Diane Atkinson and Colin Sibley --
In utero imaging of the placenta / Penny Gowland --
Animal models of fetal growth restriction and cerebral compromise / Donald Peebles --
Placental programming leading to mental ill-health: fetal growth and schizophrenia / Kathryn Abel and Matthew Allin --
Conclusions-the placenta and neurodisability / Stephen D'Souza.
Series Title: Clinics in developmental medicine, no. 169.
Responsibility: edited by Philip Baker and Colin Sibley.
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Abstract:

Contains detailed accounts of placental lesions that affect normal structure and function to show how impaired placental function may contribute to deviant fetal growth and altered brain development.

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