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Works: 14 works in 26 publications in 2 languages and 101 library holdings
Genres: Biography  Pictorial works  Prescriptions, formulae, receipts, etc  History 
Roles: Author
Publication Timeline
Publications about 马吉祥
Publications by 马吉祥
Most widely held works by 马吉祥
Zang chuan fo jiao gao seng zhuan lue by Guiming Yang( Book )
5 editions published in 1992 in Chinese and held by 39 libraries worldwide
Zhongguo Zang chuan fo jiao bai miao tu ji ( Book )
3 editions published in 1999 in Chinese and held by 16 libraries worldwide
Zhongguo Zang mi sheng xiang jie shuo by Jixiang Ma( Book )
3 editions published in 1998 in Chinese and held by 13 libraries worldwide
Zang chuan fo jiao sheng xiang jie shuo by Aluo Renqingjiebo( Book )
2 editions published in 2013 in Chinese and held by 10 libraries worldwide
I love you, my bunnies by Delisike( Book )
2 editions published between 2009 and 2010 in Chinese and held by 7 libraries worldwide
Shi yong lin zheng Zhong yao shou ce = Shiyong linzheng Zhong yao shouce ( Book )
2 editions published in 1996 in Chinese and held by 5 libraries worldwide
Thumper finds a friend by Delisike( Book )
1 edition published in 2010 in Chinese and held by 3 libraries worldwide
Exploration of the molecular structure of escherichia coli cytochrome bo ubiquinol oxidase by genetic approach by Jixiang Ma( Archival Material )
2 editions published in 1995 in English and held by 2 libraries worldwide
Cytochrome bo ubiquinol oxidase is one of the two terminal ubiquinol oxidases in the aerobic respiratory chain of Escherichia coli. By deleting the intergenic region between the cyoA and cyoB and one base in the overlapping sequence between cyoB and cyoC, in-frame fusions are made between all three subunits (II-I-III), the resulting gene product still assembles as part of a functional oxidase. The fused subunit (II-I-III) contains 22 transmembrane spans. These data support the previously proposed topology of the subunits. The purified cytochrome bo oxidase contains four subunits. The observed molecular weight of subunit II by mass spectroscopy is considerably less than the calculated value from the deduced amino acid sequence of its corresponding gene cyoA. The similarity of the N-terminal signal sequence of subunit II with those of known lipoproteins suggest that it is modified by lipids. This is proved by demonstrating that subunit II incorporates radioactive palmitic acid. A series of partial deletion mutants on subunit II have been constructed. Removal of the redundant C-terminal tail has no influence on the structure and function of the oxidase, whereas other deletion mutants are totally non-functional. Both the low-spin heme and the high-spin heme sites are severely disturbed, as manifested by the misincorporation of heme B into the binuclear center and the abnormal $alpha$-peak in the reduced minus oxidized difference optical spectra. Some deletion mutants have not shown any ubiquinol oxidase activity. It is likely that subunit II is involved in quinol binding. Site-directed mutagenesis has been conducted on all the highly conserved residues in subunit II and certain residues that are conserved in the quinol oxidase but not in cytochrome c oxidase. Replacement of the charged Glu89 with neutral residues gives rise to non-functional mutant oxidases. Of the total 33 site-directed mutants constructed in this work, only the mutant W136A shows an unusually high K$rmsb{M}$ value when compared to the wild-type. The data suggest that Trp136 is involved in the quinol binding. Cytochrome bo oxidase is able to incorporate heme B and heme O. Comparison of oxidases purified from various strains with different proportions of heme B and heme O has led to the conclusion that the $bosb3$-type oxidase is much more active than the $oosb3$-type oxidase, and the $bbsb3$-type is the least active form. Finally, it has been found that deletion of the genes for subunits III and IV leads to a weak interaction between subunit I and subunit II. When cyoC (subunit III) or cyoD (subunit IV) is deleted, the mutant oxidases inserted into the cytoplasmic membrane have no detectable ubiquinol-1 oxidase activity. It seems that subunit III and subunit IV are required for the assembly of a functional cytochrome bo oxidase
Chu xiong zhou ren shi zhi : 1988-2010 ( Book )
1 edition published in 2010 in Chinese and held by 1 library worldwide
Global, regional, and national comparative risk assessment of 79 behavioural, environmental and occupational, and metabolic risks or clusters of risks in 188 countries, 1990–2013: a systematic analysis for the Global Burden of Disease Study 2013 ( file )
1 edition published in 2015 in English and held by 1 library worldwide
Background: The Global Burden of Disease, Injuries, and Risk Factor study 2013 (GBD 2013) is the first of a series of annual updates of the GBD. Risk factor quantification, particularly of modifiable risk factors, can help to identify emerging threats to population health and opportunities for prevention. The GBD 2013 provides a timely opportunity to update the comparative risk assessment with new data for exposure, relative risks, and evidence on the appropriate counterfactual risk distribution. Methods: Attributable deaths, years of life lost, years lived with disability, and disability-adjusted life-years (DALYs) have been estimated for 79 risks or clusters of risks using the GBD 2010 methods. Risk–outcome pairs meeting explicit evidence criteria were assessed for 188 countries for the period 1990–2013 by age and sex using three inputs: risk exposure, relative risks, and the theoretical minimum risk exposure level (TMREL). Risks are organised into a hierarchy with blocks of behavioural, environmental and occupational, and metabolic risks at the first level of the hierarchy. The next level in the hierarchy includes nine clusters of related risks and two individual risks, with more detail provided at levels 3 and 4 of the hierarchy. Compared with GBD 2010, six new risk factors have been added: handwashing practices, occupational exposure to trichloroethylene, childhood wasting, childhood stunting, unsafe sex, and low glomerular filtration rate. For most risks, data for exposure were synthesised with a Bayesian meta-regression method, DisMod-MR 2.0, or spatial-temporal Gaussian process regression. Relative risks were based on meta-regressions of published cohort and intervention studies. Attributable burden for clusters of risks and all risks combined took into account evidence on the mediation of some risks such as high body-mass index (BMI) through other risks such as high systolic blood pressure and high cholesterol. Findings: All risks combined account for 57·2% (95% uncertainty interval [UI] 55·8–58·5) of deaths and 41·6% (40·1–43·0) of DALYs. Risks quantified account for 87·9% (86·5–89·3) of cardiovascular disease DALYs, ranging to a low of 0% for neonatal disorders and neglected tropical diseases and malaria. In terms of global DALYs in 2013, six risks or clusters of risks each caused more than 5% of DALYs: dietary risks accounting for 11·3 million deaths and 241·4 million DALYs, high systolic blood pressure for 10·4 million deaths and 208·1 million DALYs, child and maternal malnutrition for 1·7 million deaths and 176·9 million DALYs, tobacco smoke for 6·1 million deaths and 143·5 million DALYs, air pollution for 5·5 million deaths and 141·5 million DALYs, and high BMI for 4·4 million deaths and 134·0 million DALYs. Risk factor patterns vary across regions and countries and with time. In sub-Saharan Africa, the leading risk factors are child and maternal malnutrition, unsafe sex, and unsafe water, sanitation, and handwashing. In women, in nearly all countries in the Americas, north Africa, and the Middle East, and in many other high-income countries, high BMI is the leading risk factor, with high systolic blood pressure as the leading risk in most of Central and Eastern Europe and south and east Asia. For men, high systolic blood pressure or tobacco use are the leading risks in nearly all high-income countries, in north Africa and the Middle East, Europe, and Asia. For men and women, unsafe sex is the leading risk in a corridor from Kenya to South Africa. Interpretation: Behavioural, environmental and occupational, and metabolic risks can explain half of global mortality and more than one-third of global DALYs providing many opportunities for prevention. Of the larger risks, the attributable burden of high BMI has increased in the past 23 years. In view of the prominence of behavioural risk factors, behavioural and social science research on interventions for these risks should be strengthened. Many prevention and primary care policy options are available now to act on key risks. Funding: Bill & Melinda Gates Foundation
Social position and chronic conditions across the life span and risk of stroke: a life course epidemiological analysis of 22 847 American adults in ages over 50 ( Article )
1 edition published in 2012 in English and held by 1 library worldwide
Abstract : Background: Evidence is limited on the impact of childhood socioeconomic status, adulthood socioeconomic status and chronic conditions on risk of incident stroke in later life. We aimed to examine these associations using data from a nationally representative sample of the Health and Retirement Study. Methods: Stroke-free participants (n = 22 847) aged> 50 years in the Health and Retirement Study (1992-2008) were analyzed. Childhood and adulthood socioeconomic status were assessed using parental and participant's education attainments. Incident stroke was defined as self-reported first incident stroke. Results: Of the study sample, 2298 subjects experienced first incident stroke (10·06%). Cox's regression models indicate that subjects with low childhood socioeconomic status had 1·36 times higher risk (95% confidence interval: 1·18-1·57) of first incident stroke than those with high childhood socioeconomic status. There was an 8% reduction of this association after adjustment for adulthood socioeconomic status. Adults with diabetes mellitus had the highest hazard ratio (1·91, 95% confidence interval: 1·63-2·23) for incident stroke, followed by heart disease (1·69, 1·48-1·93), and then hypertension (1·56, 1·40-1·75). Significant interaction effect of childhood socioeconomic status and diabetes mellitus, and combined effects of socioeconomic status and chronic conditions on risk of incident stroke were observed. Conclusions: Both low socioeconomic status in childhood and adulthood socioeconomic status predict the risk of stroke. There are significantly combined effects of socioeconomic status and chronic conditions on the risk of stroke. Improving socioeconomic status across the life span and aggressive control of chronic conditions may play pivotal roles in the prevention of stroke development
Global, regional, and national incidence, prevalence, and years lived with disability for 301 acute and chronic diseases and injuries in 188 countries, 1990–2013: a systematic analysis for the Global Burden of Disease Study 2013 ( file )
1 edition published in 2015 in English and held by 1 library worldwide
Background: Up-to-date evidence about levels and trends in disease and injury incidence, prevalence, and years lived with disability (YLDs) is an essential input into global, regional, and national health policies. In the Global Burden of Disease Study 2013 (GBD 2013), we estimated these quantities for acute and chronic diseases and injuries for 188 countries between 1990 and 2013. Methods: Estimates were calculated for disease and injury incidence, prevalence, and YLDs using GBD 2010 methods with some important refinements. Results for incidence of acute disorders and prevalence of chronic disorders are new additions to the analysis. Key improvements include expansion to the cause and sequelae list, updated systematic reviews, use of detailed injury codes, improvements to the Bayesian meta-regression method (DisMod-MR), and use of severity splits for various causes. An index of data representativeness, showing data availability, was calculated for each cause and impairment during three periods globally and at the country level for 2013. In total, 35 620 distinct sources of data were used and documented to calculated estimates for 301 diseases and injuries and 2337 sequelae. The comorbidity simulation provides estimates for the number of sequelae, concurrently, by individuals by country, year, age, and sex. Disability weights were updated with the addition of new population-based survey data from four countries. Findings: Disease and injury were highly prevalent; only a small fraction of individuals had no sequelae. Comorbidity rose substantially with age and in absolute terms from 1990 to 2013. Incidence of acute sequelae were predominantly infectious diseases and short-term injuries, with over 2 billion cases of upper respiratory infections and diarrhoeal disease episodes in 2013, with the notable exception of tooth pain due to permanent caries with more than 200 million incident cases in 2013. Conversely, leading chronic sequelae were largely attributable to non-communicable diseases, with prevalence estimates for asymptomatic permanent caries and tension-type headache of 2·4 billion and 1·6 billion, respectively. The distribution of the number of sequelae in populations varied widely across regions, with an expected relation between age and disease prevalence. YLDs for both sexes increased from 537·6 million in 1990 to 764·8 million in 2013 due to population growth and ageing, whereas the age-standardised rate decreased little from 114·87 per 1000 people to 110·31 per 1000 people between 1990 and 2013. Leading causes of YLDs included low back pain and major depressive disorder among the top ten causes of YLDs in every country. YLD rates per person, by major cause groups, indicated the main drivers of increases were due to musculoskeletal, mental, and substance use disorders, neurological disorders, and chronic respiratory diseases; however HIV/AIDS was a notable driver of increasing YLDs in sub-Saharan Africa. Also, the proportion of disability-adjusted life years due to YLDs increased globally from 21·1% in 1990 to 31·2% in 2013. Interpretation: Ageing of the world's population is leading to a substantial increase in the numbers of individuals with sequelae of diseases and injuries. Rates of YLDs are declining much more slowly than mortality rates. The non-fatal dimensions of disease and injury will require more and more attention from health systems. The transition to non-fatal outcomes as the dominant source of burden of disease is occurring rapidly outside of sub-Saharan Africa. Our results can guide future health initiatives through examination of epidemiological trends and a better understanding of variation across countries. Funding: Bill & Melinda Gates Foundation
Shan pu fa xian de xin peng you = Thumper Finds A Friend by Driscoll( Book )
1 edition published in 2010 in Chinese and held by 1 library worldwide
Sha wu di a la bo wang guo yu fei sai er guo wang zhi jian jie by Jixiang Ma( Book )
1 edition published in 1971 in Chinese and held by 1 library worldwide
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Alternative Names
Ma, Chi-hsiang
马, 吉祥 1932-...
马吉祥, 1932-
Chinese (21)
English (5)
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