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Works: 12 works in 24 publications in 2 languages and 99 library holdings
Genres: Biography  Pictorial works  Prescriptions, formulae, receipts, etc  History 
Roles: Author
Publication Timeline
Publications about 马吉祥
Publications by 马吉祥
Most widely held works by 马吉祥
Zang chuan fo jiao gao seng zhuan lue by Guiming Yang( Book )
5 editions published in 1992 in Chinese and held by 39 libraries worldwide
Zhongguo Zang chuan fo jiao bai miao tu ji ( Book )
3 editions published in 1999 in Chinese and held by 16 libraries worldwide
Zhong guo cang mi sheng xiang jie shuo by Jixiang Ma( Book )
3 editions published in 1998 in Chinese and held by 13 libraries worldwide
Zang chuan fo jiao sheng xiang jie shuo by Aluo Renqingjiebo( Book )
2 editions published in 2013 in Chinese and held by 10 libraries worldwide
I love you, my bunnies by De li si ke( Book )
2 editions published between 2009 and 2010 in Chinese and held by 7 libraries worldwide
Shi yong lin zheng Zhong yao shou ce = Shiyong linzheng Zhong yao shouce ( Book )
2 editions published in 1996 in Chinese and held by 5 libraries worldwide
Thumper finds a friend by Delisike( Book )
1 edition published in 2010 in Chinese and held by 3 libraries worldwide
Exploration of the molecular structure of escherichia coli cytochrome bo ubiquinol oxidase by genetic approach by Jixiang Ma( Archival Material )
2 editions published in 1995 in English and held by 2 libraries worldwide
Cytochrome bo ubiquinol oxidase is one of the two terminal ubiquinol oxidases in the aerobic respiratory chain of Escherichia coli. By deleting the intergenic region between the cyoA and cyoB and one base in the overlapping sequence between cyoB and cyoC, in-frame fusions are made between all three subunits (II-I-III), the resulting gene product still assembles as part of a functional oxidase. The fused subunit (II-I-III) contains 22 transmembrane spans. These data support the previously proposed topology of the subunits. The purified cytochrome bo oxidase contains four subunits. The observed molecular weight of subunit II by mass spectroscopy is considerably less than the calculated value from the deduced amino acid sequence of its corresponding gene cyoA. The similarity of the N-terminal signal sequence of subunit II with those of known lipoproteins suggest that it is modified by lipids. This is proved by demonstrating that subunit II incorporates radioactive palmitic acid. A series of partial deletion mutants on subunit II have been constructed. Removal of the redundant C-terminal tail has no influence on the structure and function of the oxidase, whereas other deletion mutants are totally non-functional. Both the low-spin heme and the high-spin heme sites are severely disturbed, as manifested by the misincorporation of heme B into the binuclear center and the abnormal $alpha$-peak in the reduced minus oxidized difference optical spectra. Some deletion mutants have not shown any ubiquinol oxidase activity. It is likely that subunit II is involved in quinol binding. Site-directed mutagenesis has been conducted on all the highly conserved residues in subunit II and certain residues that are conserved in the quinol oxidase but not in cytochrome c oxidase. Replacement of the charged Glu89 with neutral residues gives rise to non-functional mutant oxidases. Of the total 33 site-directed mutants constructed in this work, only the mutant W136A shows an unusually high K$rmsb{M}$ value when compared to the wild-type. The data suggest that Trp136 is involved in the quinol binding. Cytochrome bo oxidase is able to incorporate heme B and heme O. Comparison of oxidases purified from various strains with different proportions of heme B and heme O has led to the conclusion that the $bosb3$-type oxidase is much more active than the $oosb3$-type oxidase, and the $bbsb3$-type is the least active form. Finally, it has been found that deletion of the genes for subunits III and IV leads to a weak interaction between subunit I and subunit II. When cyoC (subunit III) or cyoD (subunit IV) is deleted, the mutant oxidases inserted into the cytoplasmic membrane have no detectable ubiquinol-1 oxidase activity. It seems that subunit III and subunit IV are required for the assembly of a functional cytochrome bo oxidase
Social position and chronic conditions across the life span and risk of stroke: a life course epidemiological analysis of 22 847 American adults in ages over 50 ( file )
1 edition published in 2012 in English and held by 1 library worldwide
Abstract : Background: Evidence is limited on the impact of childhood socioeconomic status, adulthood socioeconomic status and chronic conditions on risk of incident stroke in later life. We aimed to examine these associations using data from a nationally representative sample of the Health and Retirement Study. Methods: Stroke‐free participants ( n  = 22 847) aged > 50 years in the Health and Retirement Study (1992–2008) were analyzed. Childhood and adulthood socioeconomic status were assessed using parental and participant's education attainments. Incident stroke was defined as self‐reported first incident stroke. Results: Of the study sample, 2298 subjects experienced first incident stroke (10·06%). Cox's regression models indicate that subjects with low childhood socioeconomic status had 1·36 times higher risk (95% confidence interval: 1·18–1·57) of first incident stroke than those with high childhood socioeconomic status. There was an 8% reduction of this association after adjustment for adulthood socioeconomic status. Adults with diabetes mellitus had the highest hazard ratio (1·91, 95% confidence interval: 1·63–2·23) for incident stroke, followed by heart disease (1·69, 1·48–1·93), and then hypertension (1·56, 1·40–1·75). Significant interaction effect of childhood socioeconomic status and diabetes mellitus, and combined effects of socioeconomic status and chronic conditions on risk of incident stroke were observed. Conclusions: Both low socioeconomic status in childhood and adulthood socioeconomic status predict the risk of stroke. There are significantly combined effects of socioeconomic status and chronic conditions on the risk of stroke. Improving socioeconomic status across the life span and aggressive control of chronic conditions may play pivotal roles in the prevention of stroke development
Sha wu di a la bo wang guo yu fei sai er guo wang zhi jian jie by Jixiang Ma( Book )
1 edition published in 1971 in Chinese and held by 1 library worldwide
Shan pu fa xian de xin peng you = Thumper Finds A Friend by Driscoll( Book )
1 edition published in 2010 in Chinese and held by 1 library worldwide
Chu xiong zhou ren shi zhi : 1988-2010 ( Book )
1 edition published in 2010 in Chinese and held by 1 library worldwide
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Alternative Names
Ma, Chi-hsiang
马, 吉祥 1932-...
马吉祥, 1932-
Chinese (21)
English (3)
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