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Horizons in cancer research. Volume 54

Author: Hiroto S Watanabe
Publisher: New York : Nova Biomedical, 2014. ©2014
Edition/Format:   eBook : Document : EnglishView all editions and formats
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Genre/Form: Electronic books
Additional Physical Format: Print version:
Horizons in cancer research. Volume 54.
New York : Nova Biomedical, ©2014
xii, 199 pages
Material Type: Document, Internet resource
Document Type: Internet Resource, Computer File
All Authors / Contributors: Hiroto S Watanabe
ISBN: 9781633213906 1633213900
OCLC Number: 884594738
Description: 1 online resource (213 pages)
Contents: HORIZONS IN CANCER RESEARCH. VOLUME 54; Library of Congress Cataloging-in-Publication Data; Contents; Preface; Chapter 1: Glioblastoma: Risk Factors, Diagnosis and Treatment Options; Abstract; Introduction; Risk Factor; Diagnosis; Treatment; Surgery; Radiation Therapy; Chemotherapy; Target Therapy; Recurrent GBM; References; Chapter 2: The Biology of the Interleukin-2 (IL-2)-Based Immunotherapies in Cancer and Autoimmune Disease; Abstract; Introduction; 1. Pleiotropic and Dose-Dependent Effects of IL-2 in Regulating Immune Responses; 1.1. Pleiotropic Effects of IL-2 Signaling. 1.2. IL-2 and the Mainteinance of Peripheral Tolerance1.3. Opposite Effects of IL-2 in Regulating T Cell Responses; 2. IL-2 Immunotherapy to Treat Autoimmune Diseases; 2.1. Genetic Defects in the IL-2 and IL-2R Genes and Development of Autoimmune Diseases; 2.2. IL-2 Therapy to Boost Treg Responses and Prevent Autoimmune Disorders; 2.3. Potential Use of IL-2-Immunotheraphy to Prevent Self-Reactive B Cell Responses; 2.4. IL-2 Deficiency and Th17 Driven Autoimmune Disorders; 3. IL-2-Based Anti-Tumor Immunotherapies; 3.1. IL-2 and Immunotherapy against Metastatic Cancer. 3.2. Combined IL-2 and Rituximab Therapy3.3. IL-2 to Help Anti-Tumor T-Cell Therapies; References; Chapter 3: OFF or ON: RNA Interference (RNAi) / RNA Activation (RNAa) for Cancer Therapeutics; Abstract; 1. Background; 2. siRNA for Cancer Therapeutics; 3. saRNA in Developing Cancer Therapeutics; 3.1. Liver Cancer; 3.2. Pancreatic Cancer; 3.3. Prostate Cancer; 3.4. Lung Cancer; 3.5. Cervical and Bladder Cancer; 4. Delivery into Tumors; 4.1. Aptamer Mediated Delivery; 4.2. Nanoparticle-Based Delivery; 4.3. Cell Penetrating Peptides (CPPs) Mediated Delivery. 5. Strategies to Improve Small RNA Based Cancer Therapeutics6. Chemical Modification; Conclusion Remarks and Future Perspectives; References; Chapter 4: Melanoma Progression to Metastatic Disease: The Role of the PI3K/AKT and Wnt/<U+00dd>-Catenin Pathways; Abstract; Section 1. PI3K/AKT Signaling Pathway and Its Components; 1.1. Overview; 1.2. Components of the PI3K Pathway; 1.2.1. PI3K Isoforms; 1.2.2. Phosphatase and Tensin Homologue Deleted on Chromosome 10 (PTEN); 1.2.3. AKT; 1.3. PI3K Pathway Activation and Downstream Targets; 1.4. PI3K in Cancer; Section 2. PI3K Pathway in Melanoma.
Responsibility: Hiroto S. Watanabe, editor.

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